Melissa L. Palmer - Publications

Peer-Reviewed Publications

Palmer, M.L., E.R. Peitzman, P.J. Maniak, G.C. Sieck, Y.S. Prakash, and S.M. O’Grady. 2011. KCa3.1 channels facilitate K+ secretion or ENaC-dependent Na+ absorption depending on apical or basolateral P2Y receptor stimulation. J. Physiol. 1;589(14):3483-3494, 2011.

Palmer, M.L., K.R. Schiller, and S.M. O’Grady. 2008. Apical SK potassium channels and Ca2+-dependent anion secretion in endometrial epithelial cells. J. Physiol. 1;586(3):717-26, 2008.

Lee, S.Y., M.L. Palmer, P.J. Maniak, S.H. Jang, R.D. Ryu, and S.M. O’Grady. 2007. P2Y receptor regulation of sodium transport in human mammary epithelial cells. Am. J. Physiol. Cell Physiol. 293(5):C1472-80, 2007.

Palmer, M.L., S.Y. Lee, P.J. Maniak, D. Carlson, S.F. Fahrenkrug, and S.M. O’Grady. 2006. Protease-activated receptor regulation of Cl- secretion in Calu-3 cells requires prostaglandin release and CFTR activation. Am. J. Physiol. Cell Physiology, 290: C1189-C1198, 2006.

Palmer, M.L., S.Y. Lee, D. Carlson, S.F. Fahrenkrug, and S.M. O’Grady. 2005. Stable knockdown of CFTR establishes a role for the channel in P2Y receptor-stimulated anion secretion. J. Cell Physiol. 206:759-770, 2005.

Palmer-Densmore, M.L., C. Deachapunya, M. Kannan, and S.M. O’Grady. 2002. UTP-dependent inhibition of Na+ absorption requires activation of PKC in endometrial epithelial Cells. J. Gen. Physiol., 120:897-906, 2002.

Deachapunya, C., M.L. Palmer-Densmore, and S.M. O’Grady. 1999. Insulin stimulates transepithelial sodium transport by activation of a protein phosphatase that increases Na-K ATPase activity in endometrial epithelial cells. J. Gen. Physiol. 114:561-574, 1999.

Palmer-Densmore, M.L., A.F. Johnson, and M.I. Sabara. 1998. Development and evaluation of an ELISA to measure antibody responses to both the nucleocapsid and spike proteins of canine coronavirus. J. Immunoassay, 19 (1):1-22, 1998.

Yule, T.D., M.B. Roth, K. Dreier, A.F. Johnson, M.L. Palmer-Densmore, K. Simmons, and R. Fanton. 1997. Canine parvovirus vaccine elicits protection from the inflammatory and clinical consequences of the disease. Vaccine, 15(6-7):720-9, 1997.